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In the TCR-HA/RIP-HA double-transgenic (dTg) murine model of autoimmune diabetes, oral administration of atorvastatin elicited a marked immunophenotypic reorientation: splenic lymphocyte populations in “protected” atorvastatin-treated mice exhibited a pronounced Th2-dominant signature, in contrast to the overt Th1 predominance observed in untreated diabetic counterparts [244]