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Multi-omics results show that ALOX15 is the main mediator of phospholipid peroxidation, acting as an “ignition point” during the ischemia stage, igniting phospholipid oxidation and promoting cell ferroptosis

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There was no statistically significant difference for preeclampsia, severe preeclampsia, preterm birth, or neonatal death incidence when comparing women receiving antioxidants and the placebo group