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91 , 94 , 95 The accelerated destruction of red cells imposes a bilirubin load on an already damaged liver, resulting in an exaggerated increase in serum bilirubin level

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It also promotes microbial hydrogen sulfide (H₂S) production, activating the vagal transient receptor potential cation channel subfamily V member 1 (TRPV1) receptor and modulating microglial polarization, thereby alleviating neuroinflammation following cerebral ischemia (Ni et al., 2022[133])